A role for von Willebrand factor in Streptococcus sanguis-induced platelet activation.

نویسندگان

  • Archibald McNicol
  • Erin Eyer
  • Elke C Jackson
  • Sara J Israels
چکیده

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HEMOSTASIS, THROMBOSIS, AND VASCULAR BIOLOGY A role for glycoprotein Ib in Streptococcus sanguis–induced platelet aggregation

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A role for glycoprotein Ib in Streptococcus sanguis-induced platelet aggregation.

Numerous studies have implicated bacteria in cardiovascular disease, but there is a paucity of information on the mechanism involved. In this study we show how the common oral bacterium Streptococcus sanguis can directly interact with platelets, resulting in activation and aggregate formation. Platelet aggregation was dependent on glycoprotein IIb/IIIa (GPIIb/IIIa) and thromboxane. Platelets co...

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OBJECTIVE The platelet receptor for von Willebrand factor, the glycoprotein Ib-IX (GPIb-IX) complex, mediates platelet adhesion at sites of vascular injury and transmits signals leading to platelet activation. von Willebrand factor/GPIb-IX interaction sequentially activates the Src family kinase Lyn (SFK), phosphoinositide 3-kinase (PI3K), and Akt, leading to activation of integrin α(IIb)β(3) a...

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Amplification of bacteria-induced platelet activation is triggered by FcγRIIA, integrin αIIbβ3, and platelet factor 4.

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عنوان ژورنال:
  • Thrombosis and haemostasis

دوره 98 6  شماره 

صفحات  -

تاریخ انتشار 2007